The rate of referrals to ENT clinics due to dysphonia is 1% and 11% of them constitute benign vocal cord lesions [4]. The underlying etiology of benign laryngeal lesions is vocal trauma due to misuse of the voice. Secondarily, smoking, allergy, and acid reflux also play a major role in the formation of diseases by causing mucosal damage [5,6]. Voice disorders due to benign laryngeal lesions negatively affect both the social and work-life of individuals, thus causing functional, physical, psychological and economic impairments in the quality of life [7]. In our study, in which we evaluated the age, gender, clinical symptoms and pathological features of the lesions in patients with benign laryngeal lesions, it was found that the majority of the patients (86%) were between the ages of 30-59. The most common symptom (89%) seen in the patients was found to be hoarseness/change in voice. According to the study, a polyp was the most common lesion in males (71%), while the nodule was found mostly in females (76%). The nodule and polyp which are among the benign laryngeal lesions and detected most frequently are similar to each other [8]. According to Marcatullio et al., Changes in the lamina propria with mechanical trauma begin earlier in vocal cord nodules compared to polyps. Polyps occur with the later stages of wound healing [9]. Generally, bilateral and small nodules in women are common in professional voice users. Too much talking, shouting, nervous, aggressive women are nodule candidates [10,11]. Studies show that women are more vulnerable to vocal disorders. Female vocal cords anatomically have a weaker tissue mass to absorb vocal trauma. Hyaluronic acid, which is found in excess in shock-absorbing places in the body and plays a major role in wound repair, is less in the vocal cord lamina propria of females [12]. In the nodules, there is edema and fibromyxoid stroma caused by epithelial hyperplasia, edema and vascular cell infiltration in the early period due to vocal trauma. With the continuing use of false voice, fibrosis in the lamina propria and thickening in the basement membrane are observed in the late period. Unlike nodules, polyps are seen more unilaterally and larger in males. In polyp, changes occur after vascular bleeding in the submucosal area due to vocal trauma as a result of irritating factors such as smoking, infections, allergy, gastroesophageal reflux, throat clearing habit, as well as misuse and abuse of the voice [13]. In their research on the etiology of polyps, Wang et al. Reported that the amount of pepsin in the cell fluid of polyps increased and laryngopharyngeal reflux also played a potential role in the development of the disease [14]. Hoarseness-changes and voice fatigue are common symptoms in polyps and nodules. In the treatment of nodules and polyps, vocal hygiene ve voice therapy which includes the elimination of wrong vocal habits and teaching correct techniques is the first choice. Endolaryngeal microsurgery is applied in nodules and polyps that are not treated for a long time and do not respond to voice therapy. To prevent recurrence, vocal hygiene should be ensured after the operation and voice therapy should also be applied to eliminate the habit of misuse voice [15-17]. Reinke's edema and vocal cord cyst are also seen in people who misuse their voices, have a history of reflux, smoke, talk too much. In Reinke edema, mucinous and gelatinous material accumulation occurs in the place known as the Reinke space between the superficial lamina propria under the mucosa. In a vocal cord cyst, an accumulation of serous or mucous fluid is formed in the Reinke space surrounded by a thin wall. Vocal cord cysts are thought to occur as a result of obstruction of the mucous gland ducts as a result of inflammation caused by abuse of voice, gastroesophageal reflux and upper respiratory tract infections, or the covering of the newly healing mucosa over the damaged epithelium after vocal trauma. Flexibility in the vocal cords decreases and vibration is forced [18-21]. Granuloma occurs with the effect of secondary trauma due to abuse of voice, chronic cough, throat clearing habit, intubation and gastroesophageal reflux. In a study showing that reflux plays an active role in granuloma formation, reflux was found in 30-76% of granuloma cases. Ulceration with desquamation in the epithelium after vocal trauma and inflammatory cell infiltration in the lamina propria, ulceration and hyperplasia in the surface epithelium, and excessive granulation tissue beneath it are observed. The voices of the patients may not be affected due to the granuloma originating from the posterior and afonary part of the vocal cord. Large granulomas can cause breathing difficulties by closing the glottic distance [22,23]. Papilloma is a benign, cauliflower-looking lesion of the larynx caused by the HPV virus. In infectious lesions, papillary or acanthotic growth pattern covered with squamous epithelium is observed. Cells contain well-differentiated squamous epithelium. Maturation is regular, Koilocytic atypia and frequent mitotic activity are seen. While the adult type occupies a limited place in the larynx and does not recur if completely surgically removed, the juvenile form manifests itself as extensive, large lesions, causing respiratory distress, does not respond to treatment, and recurs very frequently [24,25].