Patients with cirrhosis are exposed to a significant risk of parietal complications dominated by umbilical hernia (OH), inguinal hernia and eventrations. Ascites causes an increase in intra-abdominal pressure which will push the peritoneum subcutaneously, the subcutaneous peritoneal sac containing ascites is then in direct contact with the skin, which will thin [4], The frequency of these hernias in a cirrhotic compared to the general population is not well known but it is probably lower than that of the umbilical hernia. It is more common in men (>90%), bilateral in 10% of cases and more often associated with the presence of ascites [12], Clinically, it is especially wide neck inguino-scrotal hernias with bursae filled with ascites that are the most troublesome. They can be complicated by strangulation, but trophic skin disorders are almost non-existent [12]. Treatment of symptomatic or complicated forms is indicated. This treatment is all the more indicated as it improves the quality of life of patients [13]. Emergency repair is also associated with a higher risk of morbidity and mortality, justifying elective repair [5]. This surgical treatment is associated with a low postoperative risk even in patients with advanced cirrhosis [12,14]. A repair on a prosthesis is to be recommended especially since the risk of parietal or infectious complications is low [13,14]. Even without a prosthesis, a low recurrence rate (2.3%) has been reported [12]. The ascites puncture which confirms the diagnosis is performed in a sterile manner by puncture in full dullness at the union of the outer 1/3 and the inner 2/3 of the line connecting the umbilicus to the left anterior superior iliac spine, the risk of Hernia increases with the number and duration of ascites attacks, the size of the hernia increases if the ascites persists and it decreases when the ascites disappears [3]. A sudden decrease in ascites can promote strangulation of a digestive loop that remains incarcerated. In some cases, after disappearance of ascites and when the hernial ring is very narrow, a small amount of ascites may persist which is encysted subcutaneously, mimicking strangulation with non-reducible hernia, during ascites flare-ups, vascularized adhesions may occur between the greater omentum which remains in the hernial sac, with an appearance of non-reducible hernia which, although not painful, may lead to the diagnosis of pseudo hernial strangulation. These pseudo strangulations must have a scanner performed to eliminate a real strangulated hernia, Trophic disorders are observed when the ascites is irreducible and they are most often associated with impaired hepatocellular function. The lack of management of ascites after surgical treatment exposes to a high rate of recurrence of hernia. The basis of ascites treatment is medical, combining fluid restriction and diuretics, but in 10% of cases it becomes refractory to medical treatment, leading to the consideration of more aggressive treatments ranging from evacuating puncture , To avoid renal failure, it is preferable, from 3 L of ascites evacuated, to transfuse a vial of 20% albumin for each 2 L evacuated. [11]. The risk of emergency surgery in cirrhotics, the occurrence of serious complications in the event of conservative treatment and a better risk analysis through very large studies of patients lead to consideration of elective repairs if the hepatocellular function is preserved and the controlled ascites [4-7].Morbidity of about 15% similar in cirrhotics and non-cirrhotics, while the risk is higher in the event of emergency surgery in cirrhotics: mortality is seven times higher (3.8% vs. 0.5%) in case of emergency repair [5]. These results encourage broadening the indications for surgery in paucisymptomatic patients.